definition epidemiology risk, COPD - . So with your pals, please proceed Ingenuity is what you will need To win the huntthe ultimate test, Let's see your search find the very the best! A chest X-ray can show emphysema, one of the main causes of COPD. 8% of all individuals 10% age >40 6 th leading cause of death worldwide, COPD - . Contact Hours: 42.5 This nursing continuing professional development activity was approved by the American Association of Critical-Care Nurses, an accredited approver by the American Nurses Credentialing Center's Commission on Accreditation. Slide 3-. We've updated our privacy policy. COPD can lead to increased vulnerability by exclusion from participating fully in society. overview of copd recap basic knowledge update, COPD - . experiencing shortness of breath at rest or with minimal activity, such as walking from one room to another. -System-wide nurse referral bonus program - earn up to $6000 per referral. Youll also find a list of resources for more help managing your COPD. Y4! t/_:]I#k,^#?&&z,$\Te"I~GJdIn(7#,+}z/I86sbV7Nw4-"erbO2Zcl-1z~cw{+htMFj;*$RQm=@9i!emM}guOs\7+hc~#uM=iM%:-:;tt >*b/)2kc/):f3c:fc:f8W):NR#iK|bj,YT=^i31:_uWYY]Fte#0&? Log in Join. Nurse CHI Pulmonary and Sleep Medicine Jan 2022 - Present 1 year 3 months. This is one of the best free PPT templates for nursing. It is an under-diagnosed, life-threatening lung disease that interferes with normal breathing and is not fully reversible. Nursing Times [online]; 116: 4, 27-30. . Avg rating:3.0/5.0. The presentation must provide information about the incidence, prevalence, and pathophysiology of the disease/disorder to the cellular level. Acute exacerbations of chronic obstructive pulmonary disease (COPD) are a risk factor for disease deterioration, 1 and patients with frequent exacerbations have increased mortality. Explore family health practice innovations and models of care for clinical nursing practice globally. View COPD Powerpoint.pptx from NUR 395 at Ameritech College of Healthcare, Draper. It appears that you have an ad-blocker running. Many in this series of 12- to 20-page easy-to-read booklets feature interactive portions for patients and caregivers to discuss together. 28-7, Emphysema Pathophysiology Hyperinflation of alveoli Destruction of alveolar walls Destruction of alveolar capillary walls Narrowed airways Loss of lung elasticity, Emphysema Pathophysiology Two types: Centrilobular (central part of lobule) Most common Panlobular (destruction of whole lobule) Usually associated with AAT deficiency, Emphysema Pathophysiology Structural changes are: Hyperinflation of alveoli Destruction of alveolar capillary walls Narrowed, tortuous small airways Loss of lung elasticity, Emphysema Pathophysiology Small bronchioles become obstructed as a result of Mucus Smooth muscle spasm Inflammatory process Collapse of bronchiolar walls Recurrent infections production/stimulation of neutrophils and macrophages release proteolytic enzymes alveolar destruction inflammation, exudate, and edema, Emphysema Pathophysiology Elastin and collagen are destroyed Air goes into the lungs but is unable to come out on its own and remains in the lung Causes bronchioles to collapse, Emphysema Pathophysiology Trapped air hyperinflation and overdistention As more alveoli coalesce, blebs and bullae may develop Destruction of alveolar walls and capillaries reduced surface area for O2 diffusion Compensation is done by increasing respiratory rate to increase alveolar ventilation Hypoxemia usually develops late in disease, Emphysema Clinical Manifestations Dyspnea Progresses in severity Patient will first complain of dyspnea on exertion and progress to interfering with ADLs and rest, Emphysema Clinical Manifestations Minimal coughing with no to small amounts of sputum Overdistention of alveoli causes diaphragm to flatten and AP diameter to increase, Emphysema Clinical Manifestations Patient becomes chest breather, relying on accessory muscles Ribs become fixed in inspiratory position, Emphysema Clinical Manifestations Patient is underweight (despite adequate calorie intake), Chronic Bronchitis Pathophysiology Pathologic lung changes are: Hyperplasia of mucus-secreting glands in trachea and bronchi Increase in goblet cells Disappearance of cilia Chronic inflammatory changes and narrrowing of small airways Altered fxn of alveolar macrophages infections, Chronic Bronchitis Pathophysiology Chronic inflammation Primary pathologic mechanism causing changes Narrow airway lumen and reduced airflow d/t hyperplasia of mucus glands Inflammatory swelling Excess, thick mucus, Chronic Bronchitis Pathophysiology Greater resistance to airflow increases work of breathing Hypoxemia and hypercapnia develop more frequently in chronic bronchitis than emphysema, Chronic Bronchitis Pathophysiology Bronchioles are clogged with mucus and pose a physical barrier to ventilation Hypoxemia and hypercapnia d/t lack of ventilation and O2 diffusion Tendency to hypoventilate and retain CO2 Frequently patients require O2 both at rest and during exercise, Chronic Bronchitis Pathophysiology Cough is often ineffective to remove secretions because the person cannot breathe deeply enough to cause air flow distal to the secretions Bronchospasm frequently develops More common with history of smoking or asthma, Chronic Bronchitis Clinical Manifestations Earliest symptoms: Frequent, productive cough during winter Frequent respiratory infections, Chronic Bronchitis Clinical Manifestations Bronchospasm at end of paroxysms of coughing Cough Dyspnea on exertion History of smoking Normal weight or heavyset Ruddy (bluish-red) appearance d/t polycythemia (increased Hgb d/t chronic hypoxemia)) cyanosis, Chronic Bronchitis Clinical Manifestations Hypoxemia and hypercapnia Results from hypoventilation and airway resistance + problems with alveolar gas exchange, COPD Complications Pulmonary hypertension (pulmonary vessel constriction d/t alveolar hypoxia & acidosis) Cor pulmonale (Rt heart hypertrophy + RV failure) Pneumonia Acute Respiratory Failure, COPD Diagnostic Studies Chest x-rays early in the disease may not show abnormalities History and physical exam Pulmonary function studies reduced FEV1/FVC and residual volume and total lung capacity, COPD Diagnostic Studies ABGs PaO2 PaCO2 (especially in chronic bronchitis) pH (especially in chronic bronchitis) Bicarbonate level found in late stages COPD, COPD Collaborative Care Smoking cessation Most significant factor in slowing the progression of the disease, COPD Collaborative Care: Drug Therapy Bronchodilators as maintenance therapy -adrenergic agonists (e.g. Appointments 216.444.6503. Objective data is also assessed. COPD Chronic Obstructive Pulmonary Disease or Chronic Obstructive lung disease. COPD (CHRONIC PULMONARY OBSTRUCTIVE DISEASE) by SUKHERA. Each of the nine Guides for Better Living focuses on one important aspect of life with COPD. Goal: Develop a PowerPoint presentation on a cardiovascular/pulmonary disorder/disease discussed in the McCance text. We've updated our privacy policy. Sadness, tiredness, hopelessness and helplessness, social withdrawal and interpersonal problems, sleep and eating problems. Awareness and compliance with Guidelines for the management of Stable COPD in Current Outpatient practices and patients' knowledge of COPD - Active Detection of Chronic Obstructive Pulmonary Disease and Asthma in the General Population Vol 158. pp 1730 1738, 1998. Nursing Management Nursing Implementation Ambulatory and Home Care Discourage moving to places above 4000 ft. Looks like youve clipped this slide to already. Muhammad Arslan Yasin Sukhera. The NCCN 2023 Nursing Program: Advancing Oncology Nursing will provide oncology nurses with comprehensive and clinically relevant information to optimize patient education and care. . Presentation on Obesity Name Course Date Introduction Obesity is the excess of . Asthma patients can also develop chronic (not fully reversible) airflow obstruction. COPD Foundation Guides for Better Living are comprehensive educational booklets for use by individuals with COPD and their families, pulmonary rehabilitation programs, COPD support groups, and health care facilities. pulmonary edema; North Carolina State University . all you wanted to know about copd but were afraid to ask. or chronic bronchitis that was dr bruce davies. The SlideShare family just got bigger. Hypertensionor high blood pressurecan happen steadily over long periods of time and have no clear cause, called primary hypertension,. It has a simple style, with a white background and light blue waves and . Most patients have elements of each. Barnett, M. (2008). MDA, Interventional Pulmonary Course 2023, 2/23/2023 7:00:00 AM - 2/24/2023 4:45:00 PM, The activity is designed to review the current concepts and recent advances in interventional pulmonology as it pertains to the cancer patient, and to introduce new technologic advances that are poised to revolutionize the diagnosis, staging, and management of lung cancer. Scavenger Hunt Week 3, Choice 1 Here finally is your chance to see The many facets of creativity; Theories, presentations, lessons & such Without reality are nothing much. Its important for individuals with COPD to stay healthy and enjoy their favorite activities. Enjoy access to millions of ebooks, audiobooks, magazines, and more from Scribd. Chronic Obstructive -Partner with the Center for Education, Simulation, and Innovation to develop patient scenarios that foster the development of critical thinking skills. Free access to premium services like Tuneln, Mubi and more. It has a simple style, with a white background and light blue waves and lines, which convey elegance and serenity. Sputum gram stain and culture (for COLD exacerbation), Imaging Chest radiography Emphysema: obvious bullae, paucity of parenchymal markings, or hyperlucency Hyperinflation: increased lung volumes, flattening of diaphragm Does not indicate chronicity of changes Chest CT Definitive test for establishing the diagnosis of emphysema, but not necessary to make the diagnosis, Diagnostic Procedures Pulmonary function tests/spirometry Chronically reduced ratio of FEV1 to forced vital capacity (FVC) In contrast to asthma, the reduced FEV1 in COLD seldom shows large responses (>30%) to inhaled bronchodilators, although improvements up to 15% are common. Tachypnea Accessory respiratory muscle use, COPD Hallmark symptom - Dyspnea Chronic productive cough Minor hemoptysis pink puffer blue bloater. (Chronic Obstructive Pulmonary Disease) COPD Chronic Bronchitis Emphysema, Definition A disease state characterized by airflow limitation that is not fully reversible Conditions include: Emphysema: (anatomically defined condition characterized by destruction and enlargement of the lung alveoli) Chronic bronchitis: clinically defined condition with chronic cough and phlegm Small-airways disease: condition in which small bronchioles are narrowed. feeling excessively sleepy or confused. Presentations will focus on current and critical issues to provide oncology nurses with practical information that can be implemented in the practice setting. Medications and treatments with things like respiratory exercise devices and inhalers can help you enjoy many good years with COPD. Both have ingested substantial amounts of barbiturates and diazepam. If you are preparing a presentation about it you can use this Slidesgo proposal. Clipping is a handy way to collect important slides you want to go back to later. The air sacs fill up with air, like a small balloon. The presentation must educate advanced practice nurses on assessment and . And/or alison boland str respiratory medicine. Epidemiology Fourth leading cause of death in the U.S. Affects > 16 million persons in the U.S. Global Initiative for Chronic Obstructive Lung Disease (GOLD) estimates suggest that chronic obstructive lung disease (COLD) will increase from the sixth to the third most common cause of death worldwide by 2020. Download Presentation. The NEW! ( not fully reversible, tiredness, hopelessness and helplessness, social withdrawal and interpersonal problems, Sleep and problems... & gt ; 40 6 th leading cause of death in the U.S, with a background. And serenity hypertension, nursing Times [ online ] ; 116: 4,.. 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